Tuesday, July 30, 2013

What oral compound can be mixed with dbol for the best muscle gain and strength ?

What oral compound can be mixed with d-bol for the best muscle gain and strength ?

We run question of the day again, this time for www.outlawmuscle.com members.

See what they said.
Please post your comments below.

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Anadrol, because they will have a synergetic effect upon eachother. Anadrol is what i use to get my strength up and dbol is what i use to get my protein synthesis going..So the anadrol gets me lifting crazy heavy weight to tear muscle tissue, then the dbol builds that shit right back.It's a perfect mix.
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I'm assuming you mean running only dbol and one other oral compound?
I would say that proviron wouldnt be a bad choice.
Rather than adding another powerful oral that may be hard on your liver it's relatively mild but will maximize the quality of gains from the Dbol.
The proviron will act as an androgen while hardening up your gains. It will also suppress the amount of estrogen in your body, limit the sides and bloat while freeing up your test.
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If i had to stack two orals with no test I would have to go with Dbol AND ANAVAR.
wk1-6 dbol 30mg ed
wk1-10 anavar 50mg ed

Dbol and var would give crazy powder to you.
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for quality size and strength i would go with Dbol/primo acetate in oral form of course.

the primo will help alleviate some of the problems common with dbol like bloat and gyno. primo itself can act as a mild anti-e, great gains in strength, enhance other drugs, and some fat loss depending on diet. overall you should see some nice quality gains with this combination as they work together and leave you looking like a bloated pig.
------------------------------------------methyltrienolone.
since you didn't ask for healthiest, or least amount of sides..

while dbol is an awesome, potent oral with potential for great gains in size and mass, it is known to bloat and have many estrogenic sides (water retention, added fat mass) and the strength gains aren't off the charts, although they are worthy of mention. Enter methyltrienolone. Much like the compound it is derived from (trenbolone), it does not aromatize like d-bol, and its properties produce tremendous Strength gains while adding pounds of extreme-quality, retainable muscle mass to your frame.
------------------------------------------dbols (a better answer but vague answer is to stacked with a class 2 steroid.)

This was a theory behind stacking of class 1 and class 2 compounds. (mixed approval theory)

Within theory anavar or var is considered to be class 1, when stacked with a class 2, you get a synergistic combo.
------------------------------------------How about oral Winstrol because it has higher androgenic effects to counter balance D-Bol's low androgenic ability.
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Sunday, July 28, 2013

Oxymetholone


Oxymetholone (Anadrol) by www.apxlabs.com comes in a pouch of 50 tabs. Each tab is 50mg. There is a hologram on the pouch.

By Anthony Roberts

Oxymetholone (Anadrol) is perhaps second only to Dianabol (methandrostenolone) in importance as an oral anabolic in bodybuilding. This is due to its undoubted efficacy.

Like methandrostenolone, oxymetholone does not bind strongly to the androgen receptor (AR). Most of the anabolism it provides is therefore presumably via non-AR-mediated effects.

When using either Anadrol or Dianabol at maximum recommended dose, adding more of the other seems to yield no additional effect. For this reason, generally one drug or the other is chosen, rather than taking both at the same time.

In contrast, combining Anadrol with even a very high dose of a Class I steroid such as trenbolone, oxandrolone, or Primobolan yields a large increase in effect Oxymetholone does not aromatize: there is no conversion to estrogen.

Contrary to what many bodybuilders expect of it, the drug can be mild in terms of side effects when no aromatizing steroids are present.

Nonetheless, when oxymetholone is used in a cycle yielding high estrogen levels, it is notorious for worsening apparently-estrogenic symptoms. This may be from its producing progestagenic symptoms which are easily confused as being estrogenic; from altering estrogen metabolism; by upregulating aromatase; or perhaps by increasing prolactin. The actual cause is not proven.

There is some indirect evidence that this may be from progestagenic activity, as in some cases concurrent use of stanozolol (Winstrol), which has some anti-progestagenic effect, can avoid the problem. Some have also reported cabergoline (Dostinex) usage, which reduces prolactin, to yield a remedy.

It is primarily in the context of usage in high-estrogen circumstances that Anadrol has earned a reputation of being a harsh drug. An example such use would be combination with high-dose testosterone without an aromatase inhibitor. Most do not find it harsh when there are no concurrent problems with high estrogen.

Regardless of being non-aromatizable, in those who have developed gynecomastia already Anadrol can be an aggravating agent, even with estrogen levels kept normal. It may also be a causative agent.

For those with gynecomastia problems who are considering Anadrol and are uncertain of their response to it, rather than rely on cabergoline and/or Winstrol for protection,

I recommmend instead using Dianabol with an aromatase inhibitor or a selective estrogen receptor modulator (SERM) such as Clomid or Nolvadex.

Those not having pre-existing gynecomastia generally do well with Anadrol provided estrogen levels are not allowed to become excessive during the cycle. The above protective measures generally will not be required.

It is not unusual for a first time user to do quite well on an oxymetholone-only cycle, but the most effective use comes with stacking with a Class I steroid. Typical use is 50-150 mg/day, which is best divided into several doses per day. Higher daily doses have been used but it is not at all clear that there is any further anabolic effect from doing this. It seems to me that there is not.

When used alone, testosterone production may not completely suppressed, as there seems no indication that estrogen levels drop abnormally low, as occurs with completely suppressed testosterone production. If stacking with a non-aromatizing injectable, some amount of testosterone or other aromatizable steroid should also be used; or alternately the testosterone can be provided via low-dose HCG usage. If injectable testosterone is used, even 100 mg/week is sufficient for this purpose.

Because oxymetholone is 17-alkylated, it is stressful to the liver. It is better to limit use to no more than 6 weeks before taking a break of at least equal length.

While I cannot recommend anabolic steroid use for women at all, contrary to what many expect based on perception of men with regard to entirely differing side effects,

Anadrol has been shown medically to have a low rate of virilization at doses considerably higher than needed for non-extreme female bodybuilding or strength training. A total dosage of 25 mg/day is only half of the minimum medical dose ever routinely used, but is remarkably effective for muscle anabolism in women. Even 12.5 mg/day can be quite effective. As with any female use of oral anabolic steroids, divided doses across the day are probably safer than single-dose use for given total dosage per day, as peak levels will not be as high.

Friday, July 26, 2013

Phenom Pharmacy

New UG Lab from East Europe. They have got 6 products so far.

Stanzolol 1ml/100mg (water base)
Testosterone Propionate 1ml/100mg
Nandrolone Decanoate 1ml/250mg
Trenbolone Blend(acetate-50, hexa-50, enanthate-50) 1ml/150mg
Testosterone Blen (propionate-100, enanthate-150, cypionate-100) 1ml/ 350mg
X Blend (boldenone-150,drostanolone enanthate-150,trenbolone enantahte-100) 1ml/400mg

Nice design, silk printing, transparent box. Let's see how they progress.


Wednesday, July 24, 2013

Nolvadex vs Clomid

Tamofixen (Nolvadex) is manufactured by www.APXlabs.com. Each pouch contains 60 tabs, each tab is 20mg.

by William Llewellyn

I have received a lot of heat lately about my preference for Nolvadex over Clomid, which I hold for all purposes of use (in the bodybuilding world anyway); as an anti-estrogen, an HDL (good) cholesterol-supporting drug, and as a testosterone-stimulating compound. Most people use Nolvadex to combat gynecomastia over Clomid anyway, so that is an easy sell. And for cholesterol, well, most bodybuilders unfortunately pay little attention to this important issue, so by way of disinterest, another easy opinion to discuss. But when it comes to using Nolvadex for increasing endogenous testosterone release, bodybuilders just do not want to hear it. They only seem to want Clomid. I can only guess that this is based on a long rooted misunderstanding of the actions of the two drugs. In this article I would therefore like to discuss the specifics for these two agents, and explain clearly the usefulness of Nolvadex for the specific purpose of increasing testosterone production.

Clomid and Nolvadex

I am not sure how Clomid and Nolvadex became so separated in the minds of bodybuilders. They certainly should not be. Clomid and Nolvadex are both anti-estrogens belonging to the same group of triphenylethylene compounds. They are structurally related and specifically classified as selective estrogen receptor modulators (SERMs) with mixed agonistic and antagonistic properties. This means that in certain tissues they can block the effects of estrogen, by altering the binding capacity of the receptor, while in others they can act as actual estrogens, activating the receptor. In men, both of these drugs act as anti-estrogens in their capacity to oppose the negative feedback of estrogens on the hypothalamus and stimulate the heightened release of GnRH (Gonadotropin Releasing Hormone). lh - leutenizing hormone - output by the pituitary will be increased as a result, which in turn can increase the level of testosterone by the testes. Both drugs do this, but for some reason bodybuilders persist in thinking that Clomid is the only drug good at stimulating testosterone. What you will find with a little investigation however is that not only is Nolvadex useful for the same purpose, it should actually be the preferred agent of the two.

Pituitary Sensitivity to GnRH

Studies conducted in the late 1970's at the University of Ghent in Belgium make clear the advantages of using Nolvadex instead of Clomid for increasing testosterone levels (1). Here, researchers looked the effects of Nolvadex and Clomid on the endocrine profiles of normal men, as well as those suffering from low sperm counts (oligospermia). For our purposes, the results of these drugs on hormonally normal men are obviously the most relevant. What was found, just in the early parts of the study, was quite enlightening. Nolvadex, used for 10 days at a dosage of 20mg daily, increased serum testosterone levels to 142% of baseline, which was on par with the effect of 150mg of Clomid daily for the same duration (the testosterone increase was slightly, but not significantly, better for Clomid). We must remember though that this is the effect of three 50mg tablets of Clomid. With the price of both a 50mg Clomid and 20mg Nolvadex typically very similar, we are already seeing a cost vs. results discrepancy forming that strongly favors the Nolvadex side.

But something more interesting is happening. Researchers were also conducting GnRH stimulation tests before and after various points of treatment with Nolvadex and Clomid, and the two drugs had markedly different results. These tests involved infusing patients with 100mcg of GnRH and measuring the output of pituitary lh - leutenizing hormone - in response. The focus of this test is to see how sensitive the pituitary is to Gonadotropin Releasing Hormone. The more sensitive the pituitary, the more lh - leutenizing hormone - will be released. The tests showed that after ten days of treatment with Nolvadex, pituitary sensitivity to GnRH increased slightly compared to pre-treated values. This is contrast to 10 days of treatment with 150mg Clomid, which was shown to consistently DECREASE pituitary sensitivity to GnRH (more lh - leutenizing hormone - was released before treatment). As the study with Nolvadex progresses to 6 weeks, pituitary sensitivity to GnRH was significantly higher than pre-treated or 10-day levels. At this point the same 20mg dosage was also raising testosterone and lh - leutenizing hormone - levels to an average of 183% and 172% of base values, respectively, which again is measurably higher than what was noted 10 days into therapy. Within 10 days of treatment Clomid is already exerting an effect that is causing the pituitary to become slightly desensitized to GnRH, while prolonged use of Nolvadex serves only to increase pituitary sensitivity to this hormone. That is not to say Clomid won't increase testosterone if taken for the same 6 week time period. Quite the opposite is true. But we are, however, noticing an advantage in Nolvadex.


Clomiphene (Clomid) is manufactured by www.APXlabs.com. Each pouch contains 20 tabs, each tab is 50mg.

The Estrogen Clomid

The above discrepancies are likely explained by differences in the estrogenic nature of the two compounds. The researchers' clearly support this theory when commenting in their paper, "The difference in response might be attributable to the weak intrinsic estrogenic effect of Clomid, which in this study manifested itself by an increase in transcortin and testosterone/estradiol-binding globulin [sex hormone binding globulin ] levels; this increase was not observed after Tamoxifen treatment". In reviewing other theories later in the paper, such as interference by increased androgen or estrogen levels, they persist in noting that increases in these hormones were similar with both drug treatments, and state that," ?a role of the intrinsic estrogenic activity of Clomid which is practically absent in Tamoxifen seems the most probable explanation".

Although these two are related anti-estrogens, they appear to act very differently at different sites of action. Nolvadex seems to be strongly anti-estrogenic at both the hypothalamus and pituitary, which is in contrast to Clomid, which although a strong anti-estrogen at the hypothalamus, seems to exhibit weak estrogenic activity at the pituitary. To find further support for this we can look at an in-vitro animal study published in the American Journal of Physiology in February 1981 (2). This paper looks at the effects of Clomid and Nolvadex on the GnRH stimulated release of lh - leutenizing hormone - from cultured rat pituitary cells. In this paper, it was noted that incubating cells with Clomid had a direct estrogenic effect on cultured pituitary cell sensitivity, exerting a weaker but still significant effect compared to estradiol. Nolvadex on the other hand did not have any significant effect on lh - leutenizing hormone - response. Furthermore it mildly blocked the effects of estrogen when both were incubated in the same culture.

Conclusion

To summarize the above research succinctly, Nolvadex is the more purely anti-estrogenic of the two drugs, at least where the hpta - hypothalamic-pituitary-testicular axis - (Hypothalamic-Pituitary-Testicular Axis) is concerned. This fact enables Nolvadex to offer the male bodybuilder certain advantages over Clomid. This is especially true at times when we are looking to restore a balanced hpta - hypothalamic-pituitary-testicular axis - , and would not want to desensitize the pituitary to GnRH. This could perhaps slow recovery to some extent, as the pituitary would require higher amounts of hypothalamic GnRH in the presence of Clomid in order to get the same level of lh - leutenizing hormone - stimulation.

Nolvadex also seems preferred from long-term use, for those who find anti-estrogens effective enough at raising testosterone levels to warrant using as anabolics. Here Nolvadex would seem to provide a better and more stable increase in testosterone levels, and likely will offer a similar or greater effect than Clomid for considerably less money. The potential rise in sex hormone binding globulin levels with Clomid, supported by other research (3), is also cause for concern, as this might work to allow for comparably less free active testosterone compared to Nolvadex as well. Ultimately both drugs are effective anti-estrogens for the prevention of gynecomastia and elevation of endogenous testosterone.

Monday, July 22, 2013

Cyber Laboratories

New UG Lab from UK. Very nice pics. No feedback so far.





Saturday, July 20, 2013

Fake Pregnyl (hcg)

No doubt that Organon is legit company but this product is fake. Stay away !



Thursday, July 18, 2013

Fortis Testosterone Propionate

Not much to say about Fortis Lab. They use ink printer to make up labels, it should answer your questions...





Tuesday, July 16, 2013

Fake Russian Dbol

Just a reminder, in resposne to some emails we have received, that Russian Dbol being sold in bottles with English writing is counterfeit. There seems to be some confusion about this product among some of the readers. The only legit Russian Dbols available these days come in blister packs. Sadly, some sources (let's don't be afraid to call them scammers) carry this counterfeit and try to convince people that it is real. Feedback from those using this dbol is very bad and our advice is to stay away and stick to something real, like Jelfa Dbol.

Sunday, July 14, 2013

Lifin (finasteridum)

Lifin (Finasteridum) is manufactured by pharmaceutical company Farmacon in Poland. . Each tab is 5mg, 4 blisters, each blister is 7 tabs. Safe buy.

Finasteridum is a medication commonly known by the generic name Finasteride. It is also marketed globally under the names Chibro-Proscar, Finast, Finasterida, Finastid, Fincar, Finpecia, Propecia, Propeshia, Proscar, Prostide as well as generic Finasteride.

Finasteridum (Finasteride) is an androgen hormone inhibitor used to treat male pattern hair loss IN MEN ONLY. A doctor may prescribe Finasteridum (Finasteride) for additional conditions.